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In this context, the present study was conducted to systematically investigate potential targets and pathogenic pathways of DEHP-induced BRCA specific to Homo sapiens via network toxicology and molecular docking strategies

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Abstract The incretins glucose-dependent insulinotropic polypeptide (GIP) and glucagon-like peptide 1 (GLP-1) mediate insulin responses that are proportionate to nutrient intake to facilitate glucose tolerance 1